A team of researchers studying the protein that, when defective or absent, causes cystic fibrosis (CF) has made an important discovery about how that protein is normally controlled and under what circumstances it might go awry. "Understanding the regulation of salt transport in normal cells is critical for the development of new therapies for diseases, like CF, that disrupt salt movements across cell borders," said Jeng-Haur Chen, a postdoctoral researcher at the University of Iowa Carver College of Medicine and the lead author on a paper to be published in the Dec...
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